During the next 20 years, the plan for China and India is basically to catch up to the U.S.—to get 19th century plumbing and 20th century railroads. That is not […]

There has been lots of innovation recently in areas without regulation—heavily regulated fields like transportation, health care, and energy have been much slower to progress.

The venture capitalist agrees with Bill Gates that the free market often fails to encourage long-term innovation. But governments are not the solution.

Positive psychologists have found a series of scientifically-proven ways for you live a happier life.

Genetic testing is advancing rapidly, and we can now find out our risk factor for developing Alzheimer’s. But without a cure or treatment available, what’s the point?

Studies have shown that you can boost brainpower and brain resilience with vitamins and exercise, and by not smoking. But lifestyle choices alone can’t prevent Alzheimer’s?

Alzheimer’s starts in one area and spreads all over the brain, like an infection. Does this mean that it’s possible to develop a vaccine?

Genes such as ApoE4 may signal a risk factor for Alzheimer’s. But how do we separate risk factor from an unalterable sentence for the disease?

Amyloid plaques and neurofibrillary tangles inside the brain are the best explanation we have for how Alzheimer’s develops.

Mental decline, on some level, is inseparable from aging. With more people living longer, does this mean everyone will eventually get Alzheimer’s?

One of the most robust environmental risk factors identified for Alzheimer’s disease is traumatic brain injury. This is having repercussions for those returning from fighting in Iraq and Afghanistan.

Amyloid buildup in the brain is a key trigger in Alzheimer’s disease, but some people with this plaque live their entire lives without developing the disease.

Are women and African-Americans at a higher risk for Alzheimer’s—as some data suggests—or are there other factors in play?

For much of the past 100 years, little was known about Alzheimer’s disease and dementia. But recent research is revealing the neurotransmitter and genetic defects behind the disease.

Genetic testing is advancing rapidly, and we can now find out our risk factor for developing Alzheimer’s. But without a cure or treatment available, what’s the point?

Studies have shown that you can boost brainpower and brain resilience with vitamins and exercise, and by not smoking. But lifestyle choices alone can’t prevent Alzheimer’s?

Alzheimer’s starts in one area and spreads all over the brain, like an infection. Does this mean that it’s possible to develop a vaccine?

Genes such as ApoE4 may signal a risk factor for Alzheimer’s. But how do we separate risk factor from an unalterable sentence for the disease?

Amyloid plaques and neurofibrillary tangles inside the brain are the best explanation we have for how Alzheimer’s develops.

Mental decline, on some level, is inseparable from aging. With more people living longer, does this mean everyone will eventually get Alzheimer’s?

One of the most robust environmental risk factors identified for Alzheimer’s disease is traumatic brain injury. This is having repercussions for those returning from fighting in Iraq and Afghanistan.

Amyloid buildup in the brain is a key trigger in Alzheimer’s disease, but some people with this plaque live their entire lives without developing the disease.

Are women and African-Americans at a higher risk for Alzheimer’s—as some data suggests—or are there other factors in play?

For much of the past 100 years, little was known about Alzheimer’s disease and dementia. But recent research is revealing the neurotransmitter and genetic defects behind the disease.

Genetic testing is advancing rapidly, and we can now find out our risk factor for developing Alzheimer’s. But without a cure or treatment available, what’s the point?

Studies have shown that you can boost brainpower and brain resilience with vitamins and exercise, and by not smoking. But lifestyle choices alone can’t prevent Alzheimer’s?

Alzheimer’s starts in one area and spreads all over the brain, like an infection. Does this mean that it’s possible to develop a vaccine?

Genes such as ApoE4 may signal a risk factor for Alzheimer’s. But how do we separate risk factor from an unalterable sentence for the disease?